PSA infarction after endovascular surgery for VAD seems not to be a rare potential complication. On univariate analysis, the variables of posterior inferior cerebellar artery-involved-type, PO, and intraprocedural proximal flow arrest were significantly correlated with occurrence of PSA infarction. /rebates/&252fwallenberg-pica-syndrome-step-1. All 3 symptomatic SCIs were PSA infarction.
Periprocedural symptomatic infarction was detected in 4 of the 21 cases (19%): 3 SCIs and 1 lower medulla infarction, after 1 stent-assisted coil embolization and 3 PO. The endovascular surgical method was internal trapping in 10 cases, stent-assisted coil embolization in 8 cases, and proximal occlusion (PO) in 3 cases. Thirteen cases were unruptured aneurysms, and 8, ruptured aneurysms. The syndrome results from infarction of the medulla by vertebral artery thrombosis or dissection that may also produce occlusion of the opening to the posterior inferior cerebellar artery. The blood supply to the lateral medulla is the posterior inferior cerebellar artery. The lateral medullary syndrome, also known as Wallenbergs syndrome, is the prototype lesion involving the nuclei of cranial nerves IX and X. The variables of aneurysmal location, status, intra-aneurysmal thrombosis, antithrombotic therapy, and endovascular procedure were investigated in relation to the occurrence of spinal cord or brain stem infarction. Wallenberg syndrome or lateral medullary infarction, is associated with the acute onset of vertigo and disequilibrium. Infarction in the region of the PSA after endovascular surgery for VADs carried out in consecutive 21 cases was investigated. To investigate PSA infarction after endovascular surgery for VAD. Although SCI in the region of the anterior spinal artery (ASA) has been reported, SCI in the region of the posterior spinal artery (PSA) is rare. Endovascular surgery for vertebral artery dissections (VADs) carries the risk of spinal cord infarction (SCI).
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